Amos CI

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Genome-wide association meta-analysis identifies pleiotropic risk loci for aerodigestive squamous cell cancers
Immune-mediated genetic pathways resulting in pulmonary function impairment increase lung cancer susceptibility
Systematic analyses of regulatory variants in DNase I hypersensitive sites identified two novel lung cancer susceptibility loci
Elevated platelet count appears to be causally associated with increased risk of lung cancer: A Mendelian randomization analysis
Lung cancer risk in never-smokers of European descent is associated With genetic variation in the 5p15.33 TERT-CLPTM1Ll region
Genetic interaction analysis among oncogenesis-related genes revealed novel genes and networks in lung cancer development
Fine mapping of MHC region in lung cancer highlights independent susceptibility loci by ethnicity
Genetic modifiers of radon-induced lung cancer risk: A genome-wide interaction study in former uranium miners
Mendelian Randomization and mediation analysis of leukocyte telomere length and risk of lung and head and neck cancers
Genome-wide interaction study of smoking behavior and non-small cell lung cancer risk in Caucasian population
Pleiotropy of genetic variants on obesity and smoking phenotypes: Results from the Oncoarray Project of The International Lung Cancer Consortium
Obesity, metabolic factors and risk of different histological types of lung cancer: A Mendelian randomization study
Abstract 2292: Lung function and lung cancer risk: A Mendelian randomization study of UK Biobank cohort and the International Lung Cancer Consortium
Genetic risk can be decreased: Quitting smoking decreases and delays lung cancer for smokers with high and low CHRNA5 risk genotypes — A meta-analysis
Abstract 817: Mendelian randomization and mediation analysis of 5p15.33, telomere length and lung cancer risk
CHRNA5 risk variant predicts delayed smoking cessation and earlier lung cancer diagnosis—A meta-analysis